Neuronal Apoptosis Induced by Endoplasmic Reticulum Stress Is Regulated by ATF4–CHOP-Mediated Induction of the Bcl-2 Homology 3-Only Member PUMA | Journal of Neuroscience
Effects of endoplasmic reticulum stress on apoptosis induction in radioresistant macrophages
ER stress-induced cell death proceeds independently of the TRAIL-R2 signaling axis in pancreatic β cells | Cell Death Discovery
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect
Neuron‐specific translational control shift ensures proteostatic resilience during ER stress | The EMBO Journal
Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases | Nature Reviews Nephrology
What is Thapsigargin? Frequently Asked Questions
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology
Targeting endoplasmic reticulum stress in insulin resistance: Trends in Endocrinology & Metabolism
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text
EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download Scientific Diagram
MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites | The EMBO Journal
IJMS | Free Full-Text | An Involvement of Oxidative Stress in Endoplasmic Reticulum Stress and Its Associated Diseases
Effect of endoplasmic reticulum stress on metabolic and stress signaling and kidney-specific functions in Madin-Darby bovine kid
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1
Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78 kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and Vascular Biology
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
Figure S2. Thapsigargin-induced ER Ca 2+ release is reduced in STIM1KO... | Download Scientific Diagram
JNK activation by thapsigargin versus saturated free fatty acid (SFA).... | Download Scientific Diagram
Schematic diagram of thapsigargin-stimulated TRAIL-induced apoptosis in... | Download Scientific Diagram
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
